Quoting: Anonymous Coward 8954905 Quoting: Anonymous Coward 8951556
Not going to read 7 pages of anti-fluoride idiocy. I read the original research paper. They were studying the effectiveness of using a fluoride tracer with a CAT scan in order to image atherosclerosis. Quoting: Anonymous Coward 8954905
But whatever, some moronic website misreads a research paper, puts out a flat out incorrect headline and you morons eat it up.
You have a poor ability to abstract and deduce from evidence, not to mention a lack of rudimentary reading comprehension skill.
But the researchers did not, for they cite in their conclusion...
CONCLUSION: sodium [¹⁸F]fluoride PET/CT might be useful in the evaluation of the atherosclerotic process in major arteries, including coronary arteries. An increased fluoride uptake in coronary arteries may be associated with an increased cardiovascular risk
Yeah, I'm such a bad reader and thinker that I managed to become my class valedictorian and am read by tens of thousands of people worldwide in my field.
In any case, the increased uptake once again refers to the tracer they are using - fluoride. In other words, if someone has calcification occurring in their coronary arteries then that is probably a risk factor for heart disease.
Fluoride binds with calcium. That's why they are using it as a tracer for CAT scans. They inject you with it so it will bind with any calcification deposits and then scan you.
If the CAT scan shows increased uptake of that tracer in your coronary arteries then chances are you have calcification going on there and are at risk for heart disease.
Of course, if you weren't a grade A moron you would've read it properly instead of twisting their words to fit your fantasy.
LOL. A valedictorian of Shilltard University?
If you were so smart you would be able to take it a step forward and realize the addition of a salt to a person's bloodstream, a person who already has arterial calcification especially, will result in an INCREASE in the arterial accretions due to the affinity of various NA-FL salt compounds to lodge there, as PROVEN by that study.
In addition these compound will also shed a fluoride ion or two which will bind with calcium either on the plaque, or creating a INSOLUBLE Calcium fluoride. (see reference below)
Now while insoluble CAF2 is generally harmless, the process whereby it was created has just removed that free calcium's potential to do its proper work in regulating the nervous system, specifically by disrupting the homeostasis (proper exchange) of sodium and calcium ions across the plasma membrane of a cell. In other words, Fluoride encourages a short in the circuit by reducing free Calcium.
^ a b c I. M. Rabinowitch. Acute Fluoride Poisoning. Can Med Assoc J. 1945, 52, 345–349. 
The mechanism of toxicity involves the combination of the fluoride anion with the calcium ions in the blood to form insoluble calcium fluoride, resulting in hypocalcemia; calcium is indispensable for the function of the nervous system, and the condition can be fatal. Treatment may involve oral administration of dilute calcium hydroxide or calcium chloride to prevent further absorption, and injection of calcium gluconate to increase the calcium levels in the blood.