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Message Subject <<Advancing Bird Flu-H5N1...Now following MERS and Ebola approaching PANDEMICS>>>
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Some research indicates that the villagers of Eyam may have had some genetic protection from the bubonic plague.[3] A CCR5 gene mutation designated as "delta 32" was found in a statistically significant number, 14%, of direct descendants of the plague survivors. The Delta 32 mutation appears to be very rare. In fact, the levels of Delta 32 found in Eyam were only matched in regions of Europe that had been affected by the plague and in Americans of European origin. It has also been suggested[3] that the Delta 32 mutation, if inherited from both parents, may provide immunity to HIV/AIDS. CCR5, short for chemokine (C-C motif) receptor 5, is a chemokine receptor. ... CCR5-D32 is a genetic defect affecting the human immune system that has both harmful and beneficial effects. ...


More recent research at Scripps Research Institute disputes the hypothesis that the Delta 32 mutation provided protection against the plague, suggesting instead that it is more likely to have arisen as protection against some other disease common at the time, such as smallpox. This new hypothesis is still being tested. [4] The Scripps Research Institute, in La Jolla, California is home to notable chemists such as K. Barry Sharpless and P. G. Schultz, as well as neurobiologist Gerald Edelman, and Nobel Laureate Kurt Wurtrich. ...
I believe they are trying to find ways of reproducing this gene but understand that one of the problems have been the difficulties in O rh neg is that it can not be cloned unlike the other blood groups I find it very intriguing
 
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