BREAKING VIDEO: Assad has Unleashed Chemical Weapons in Aleppo!! (GRAPHIC) | |
BRIEF User ID: 29406202 United States 12/09/2012 09:25 AM Report Abusive Post Report Copyright Violation | |
Zeff User ID: 15574462 France 12/09/2012 09:29 AM Report Abusive Post Report Copyright Violation | roots of USA [link to www.legrandsoir.info] Berlin vs Palestine wall [link to img15.hostingpics.net] |
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Anonymous Coward User ID: 7790078 United Kingdom 12/09/2012 09:43 AM Report Abusive Post Report Copyright Violation | Nerve Agents. Nerve agents are organophosphate ester derivatives of phosphoric acid. They are generally divided into the Gagents, which in the unmodified state are volatile, and the V-agents, which tend to be more persistent. Even G-agents are capable of being thickened with various substances to increase the persistence and penetration of the intact skin. The principal nerve agents are Tabun (GA), Sarin (GB), Soman (GD), and VX. (In some countries the V-agents are known as A-agents.) The G-agents are fluorine- or cyanide-containing organophosphates. In pure form they are colorless liquids. Their solubility in water ranges from complete miscibility for GB to almost total insolubility for GD. They have a weakly fruity odor but in field concentrations are odorless. Clothing gives off G-agents for about 30 minutes after contact with vapor; consider this fact before unmasking. The V-agents are sulfur-containing organophosphorous compounds. They are oily liquids with high boiling points, low volatility, and resultant high persistency. They are primarily contact hazards. They are exceptionally toxic; the limited amount of vapor they produce is sufficient to be an inhalation hazard. They have very limited volubility in water and are hydrolyzed only minimally. V-agents affect the body in essentially the same manner as G-agents. The nerve agents are all viscous liquids, not nerve gas per se. However, the vapor pressures of the G-series nerve agents are sufficiently high for the vapors to be lethal rapidly. The volatility is a physical factor of most importance. GB is so volatile that small droplets sprayed from a plane or released from a shell exploding in the air may never reach the ground. This total volatilization means that GB is largely a vapor hazard. At the other extreme agent VX is of such low volatility that it is mainly a liquid contact hazard. Toxicity can occur from the spray falling on one’s skin or clothes and from touching surfaces on which the spray has fallen. GD is also mainly a vapor hazard, while GA can be expected to contaminate surfaces for a sufficiently long time to provide a relevant contact hazard. Thickeners added to GD increase persistence in the field. The thickened agents form large droplets that provide a greater concentration reaching the ground and a greater contact hazard than the unthickened forms. The relative volubility of these compounds in water and soil is of significance because it relates to their disposition. The ability of GB and GA to mix with water means that water could wash them off surfaces, that these agents can easily contaminate water sources, and that they will not penetrate skin as readily as the more fat-soluble agents VX and GD. G-agents spread rapidly on surfaces, such as skin; VX spreads less rapidly, and the thickened agents very slowly. The moist surfaces in the lungs absorb all the agents very well. [link to books.google.com] |
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Anonymous Coward User ID: 1405546 United States 12/09/2012 10:13 AM Report Abusive Post Report Copyright Violation | Keep Baking Soda and water handy. It neutralizes acids, works great on bee stings and other acidic neuro-toxins. Though, it isn't a listed counter. Do not assume it will save your ass, but it may help if you are on the edge of the chemical cloud or are treating someone with contact. It might save their lives. |
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DoorBert (OP) User ID: 1495371 United States 12/09/2012 10:36 AM Report Abusive Post Report Copyright Violation | Syria Says Rebels May Use Chemical Weapons From A Mysterious Factory Near Aleppo Read more: [link to pixel.newscred.com] |
Anonymous Coward User ID: 29098642 United States 12/09/2012 10:51 AM Report Abusive Post Report Copyright Violation | Not fake. more videos coming in of the actual chemical attack aftermath: [liveleak] [link to www.liveleak.com] [liveleak] [link to www.liveleak.com] |
Anonymous Coward User ID: 17487177 United States 12/09/2012 10:52 AM Report Abusive Post Report Copyright Violation | Nerve Agents. Quoting: Anderson_G Nerve agents are organophosphate ester derivatives of phosphoric acid. They are generally divided into the Gagents, which in the unmodified state are volatile, and the V-agents, which tend to be more persistent. Even G-agents are capable of being thickened with various substances to increase the persistence and penetration of the intact skin. The principal nerve agents are Tabun (GA), Sarin (GB), Soman (GD), and VX. (In some countries the V-agents are known as A-agents.) The G-agents are fluorine- or cyanide-containing organophosphates. In pure form they are colorless liquids. Their solubility in water ranges from complete miscibility for GB to almost total insolubility for GD. They have a weakly fruity odor but in field concentrations are odorless. Clothing gives off G-agents for about 30 minutes after contact with vapor; consider this fact before unmasking. The V-agents are sulfur-containing organophosphorous compounds. They are oily liquids with high boiling points, low volatility, and resultant high persistency. They are primarily contact hazards. They are exceptionally toxic; the limited amount of vapor they produce is sufficient to be an inhalation hazard. They have very limited volubility in water and are hydrolyzed only minimally. V-agents affect the body in essentially the same manner as G-agents. The nerve agents are all viscous liquids, not nerve gas per se. However, the vapor pressures of the G-series nerve agents are sufficiently high for the vapors to be lethal rapidly. The volatility is a physical factor of most importance. GB is so volatile that small droplets sprayed from a plane or released from a shell exploding in the air may never reach the ground. This total volatilization means that GB is largely a vapor hazard. At the other extreme agent VX is of such low volatility that it is mainly a liquid contact hazard. Toxicity can occur from the spray falling on one’s skin or clothes and from touching surfaces on which the spray has fallen. GD is also mainly a vapor hazard, while GA can be expected to contaminate surfaces for a sufficiently long time to provide a relevant contact hazard. Thickeners added to GD increase persistence in the field. The thickened agents form large droplets that provide a greater concentration reaching the ground and a greater contact hazard than the unthickened forms. The relative volubility of these compounds in water and soil is of significance because it relates to their disposition. The ability of GB and GA to mix with water means that water could wash them off surfaces, that these agents can easily contaminate water sources, and that they will not penetrate skin as readily as the more fat-soluble agents VX and GD. G-agents spread rapidly on surfaces, such as skin; VX spreads less rapidly, and the thickened agents very slowly. The moist surfaces in the lungs absorb all the agents very well. Both the G- and V-agents have the same physiological action on humans. They are potent inhibitors of the enzyme acetylcholinesterase (AChE), which is required for the function of many nerves and muscles in nearly every multicellular animal. Normally, AChE prevents the accumulation of acetylcholine after its release in the nervous system. Acetylcholine plays a vital role in stimulating voluntary muscles and nerve endings of the autonomic nervous system and many structures within the central nervous system. Thus, nerve agents that are cholinesterase inhibitors permit acetylcholine to accumulate at those sites, mimicking the effects of a massive release of acetylcholine. The major effects will be on skeletal muscles, parasympathetic end organs, and the central nervous system. Individuals poisoned by nerve agents may display the following symptoms: Difficulty in breathing. Drooling and excessive sweating. Nausea. Vomiting, cramps, and loss of bladder/bowel control. Twitching, jerking, and staggering. Headache, confusion, drowsiness, coma, and convulsion. The number and severity of symptoms depend on the quantity and route of entry of the nerve agent into the body. When the agent is inhaled, a prominent symptom is pinpointing of the pupils of the eyes and dimness of vision because of the reduced amount of light entering. However, if exposure has been through the skin or by ingestion of a nerve agent, the pupils may be normal or only slightly to moderately reduced in size. In this event, diagnosis must rely upon the symptoms of nerve agent poisoning other than its effects on the pupils. Exposure through the eyes produces a very rapid onset of symptoms (usually less than 2 to 3 minutes). Respiratory exposure usually results in onset of symptoms in 2 to 5 minutes; lethal doses kill in less than 15 minutes. Liquid in the eye kills nearly as rapidly as respiratory exposure. Symptoms appear much more slowly from skin absorption. Skin absorption great enough to cause death may occur in one to two hours. Respiratory lethal dosages kill in one to ten minutes, and liquid in the eye kills nearly as rapidly. Very small skin dosages sometimes cause local sweating and tremors but little other effects. Nerve agents are cumulative poisons. Repeated exposure to low concentrations, if not too far apart, will produce symptoms. Treatment of nerve agent poisoning includes use of the nerve agent antidote (atropine and 2-PAM chloride). Atropine blocks acetylcholine; 2-PAM Cl, reactivates the enzyme AChE. As time passes without treatment the binding of nerve agents to AChE “ages” and the 2-PAM Cl can no longer remove the agent. Certain agents, such as GD, that age rapidly may resist treatment if it is not prompt. Therefore, an antidote enhancer, pyridostigmine bromide (PB), is available to US forces in active theaters of operation. PB pretreatment increases the victim’s survivability when the antidote is used after exposure to nerve agents Nice copy/paste. Care to source it ? |
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Duped again! User ID: 1880435 United States 12/09/2012 11:07 AM Report Abusive Post Report Copyright Violation | |
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NSF001 User ID: 29402466 United Kingdom 12/09/2012 11:20 AM Report Abusive Post Report Copyright Violation | Not fake. more videos coming in of the actual chemical attack aftermath: Quoting: Anonymous Coward 29098642 [liveleak] [link to www.liveleak.com] [liveleak] [link to www.liveleak.com] This is not the same area as was in the first video, also if anything that would be a supher-mustard bomb, in which case the guy poking it with a stick would be suffering. It could well be napalm. I don't think so, he's up there with the others laying low, vying with those who you've traded your life to to bless your soul, |
Anonymous Coward User ID: 7790078 United Kingdom 12/09/2012 12:48 PM Report Abusive Post Report Copyright Violation | Nerve Agents. Quoting: Anderson_G Nerve agents are organophosphate ester derivatives of phosphoric acid. They are generally divided into the Gagents, which in the unmodified state are volatile, and the V-agents, which tend to be more persistent. Even G-agents are capable of being thickened with various substances to increase the persistence and penetration of the intact skin. The principal nerve agents are Tabun (GA), Sarin (GB), Soman (GD), and VX. (In some countries the V-agents are known as A-agents.) The G-agents are fluorine- or cyanide-containing organophosphates. In pure form they are colorless liquids. Their solubility in water ranges from complete miscibility for GB to almost total insolubility for GD. They have a weakly fruity odor but in field concentrations are odorless. Clothing gives off G-agents for about 30 minutes after contact with vapor; consider this fact before unmasking. The V-agents are sulfur-containing organophosphorous compounds. They are oily liquids with high boiling points, low volatility, and resultant high persistency. They are primarily contact hazards. They are exceptionally toxic; the limited amount of vapor they produce is sufficient to be an inhalation hazard. They have very limited volubility in water and are hydrolyzed only minimally. V-agents affect the body in essentially the same manner as G-agents. The nerve agents are all viscous liquids, not nerve gas per se. However, the vapor pressures of the G-series nerve agents are sufficiently high for the vapors to be lethal rapidly. The volatility is a physical factor of most importance. GB is so volatile that small droplets sprayed from a plane or released from a shell exploding in the air may never reach the ground. This total volatilization means that GB is largely a vapor hazard. At the other extreme agent VX is of such low volatility that it is mainly a liquid contact hazard. Toxicity can occur from the spray falling on one’s skin or clothes and from touching surfaces on which the spray has fallen. GD is also mainly a vapor hazard, while GA can be expected to contaminate surfaces for a sufficiently long time to provide a relevant contact hazard. Thickeners added to GD increase persistence in the field. The thickened agents form large droplets that provide a greater concentration reaching the ground and a greater contact hazard than the unthickened forms. The relative volubility of these compounds in water and soil is of significance because it relates to their disposition. The ability of GB and GA to mix with water means that water could wash them off surfaces, that these agents can easily contaminate water sources, and that they will not penetrate skin as readily as the more fat-soluble agents VX and GD. G-agents spread rapidly on surfaces, such as skin; VX spreads less rapidly, and the thickened agents very slowly. The moist surfaces in the lungs absorb all the agents very well. Both the G- and V-agents have the same physiological action on humans. They are potent inhibitors of the enzyme acetylcholinesterase (AChE), which is required for the function of many nerves and muscles in nearly every multicellular animal. Normally, AChE prevents the accumulation of acetylcholine after its release in the nervous system. Acetylcholine plays a vital role in stimulating voluntary muscles and nerve endings of the autonomic nervous system and many structures within the central nervous system. Thus, nerve agents that are cholinesterase inhibitors permit acetylcholine to accumulate at those sites, mimicking the effects of a massive release of acetylcholine. The major effects will be on skeletal muscles, parasympathetic end organs, and the central nervous system. Individuals poisoned by nerve agents may display the following symptoms: Difficulty in breathing. Drooling and excessive sweating. Nausea. Vomiting, cramps, and loss of bladder/bowel control. Twitching, jerking, and staggering. Headache, confusion, drowsiness, coma, and convulsion. The number and severity of symptoms depend on the quantity and route of entry of the nerve agent into the body. When the agent is inhaled, a prominent symptom is pinpointing of the pupils of the eyes and dimness of vision because of the reduced amount of light entering. However, if exposure has been through the skin or by ingestion of a nerve agent, the pupils may be normal or only slightly to moderately reduced in size. In this event, diagnosis must rely upon the symptoms of nerve agent poisoning other than its effects on the pupils. Exposure through the eyes produces a very rapid onset of symptoms (usually less than 2 to 3 minutes). Respiratory exposure usually results in onset of symptoms in 2 to 5 minutes; lethal doses kill in less than 15 minutes. Liquid in the eye kills nearly as rapidly as respiratory exposure. Symptoms appear much more slowly from skin absorption. Skin absorption great enough to cause death may occur in one to two hours. Respiratory lethal dosages kill in one to ten minutes, and liquid in the eye kills nearly as rapidly. Very small skin dosages sometimes cause local sweating and tremors but little other effects. Nerve agents are cumulative poisons. Repeated exposure to low concentrations, if not too far apart, will produce symptoms. Treatment of nerve agent poisoning includes use of the nerve agent antidote (atropine and 2-PAM chloride). Atropine blocks acetylcholine; 2-PAM Cl, reactivates the enzyme AChE. As time passes without treatment the binding of nerve agents to AChE “ages” and the 2-PAM Cl can no longer remove the agent. Certain agents, such as GD, that age rapidly may resist treatment if it is not prompt. Therefore, an antidote enhancer, pyridostigmine bromide (PB), is available to US forces in active theaters of operation. PB pretreatment increases the victim’s survivability when the antidote is used after exposure to nerve agents Nice copy/paste. Care to source it ? Its a field manual I got......... hold up I'll see if I can find it online...... [link to www.truthistreason.net] here we go same manual. |
Kipod User ID: 23670461 Israel 12/09/2012 01:21 PM Report Abusive Post Report Copyright Violation | |
Future Viewer User ID: 1344660 United States 12/09/2012 01:25 PM Report Abusive Post Report Copyright Violation | Fears of the worst possible scenario in Syria have been confirmed in a video released by rebels that shows victims of chemical warfare. Quoting: DoorBert An almost sure sign that Syrian President Bashar al-Assad has unleashed his chemical weapons for use against rebels is his regime’s claim on Saturday that rebels are preparing to use chemicals in their uprising. Assad systematically has accused opposition forces, which he labels as “foreign terrorists,” of carrying out attacks and murders for which his own loyalists are responsible. [link to www.israelnationalnews.com] And so it begins... As it ends... |
Anonymous Coward User ID: 21931562 United States 12/09/2012 01:32 PM Report Abusive Post Report Copyright Violation | Yep, they must have used a contingency of the FEMA, CIA, Blackwater, and CNN fake-trauma make up artists to doll up the fake news they keep pumping out. Even if they did use chemical weapons so what? The US Government uses chemicals and microwave/ELF weapons on peaceful protesters in the USSA. |
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