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Subject BREAKING! Total destruction of lungs in Ukraine fatal H!N1 Update! WHO silent on sequences. Deaths 174
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Original Message Total Destruction of Lungs in Ukraine Fatal H1N1 Cases
Recombinomics Commentary 04:22
November 9, 2009

The symptoms are observed at different stages of disease - a fever with a temperature over 38 C, cough, respiratory disorders. When cough was characterized by negligible allocation phlegm or dry unproductive cough with blotches of blood. All the patients come to hospital on average by 3-7 days of onset, were in serious condition. Period of time from onset to death averaged from 4 to 7 days. In all patients during a hospital for signs of respiratory insufficiency of various degrees, which quickly rose and manifested accelerated respiration rate, shortness of breath and effectiveness of independent breathing. X-ray studies were performed on 1-2 day hospitalization. Most patients experienced a double-headed particles of lower lung lesion, followed by a trend towards total destruction.

The above translation is from an announcement today from the Ukraine Ministry of Health describe 90 fatalities (see map) associated with community acquired pneumonia. The full report included the age distribution, which exactly match distribution reported previously by other countries for swine H1N1. 52/90 fatalities were in the age group of 19-40, followed by 26/90 in the 41-55 age group.

Coughing up blood followed the "total destruction" of lungs again gives a clinical picture of hemorrhagic pneumonia caused by H1N1 swine flu.


[link to www.recombinomics.com]

1,031,597 Influenza/ARI

52,742 Hospitalized

174 Dead

The latest update for Ukraine includes more than 1 million reported cases (see map). The fatalities have jumped from 155 to 174 and almost 53K have been hospitalized. The biggest jump in fatalities was in Lviv, where reported deaths rose from 63 to 74. However, the largest jump in cases was in Kiev, raising concerns that the infections were spreading east.

The increase in cases and deaths continue to support a genetic change in the H1N1 virus.

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