CoronaVirus really a virus? Research suggest it isn't

This is a very long, well documented and thought provoking article. There is a summary at the head of the article.

You will be going hmmm.... before you are half way through.



Trump Halts CDC Fearmongering. But Why Are Antibiotics & Not Anti-Virals Quelling The COVID-19 Coronavirus? Is It Really A Virus?

LewRockwell.com

Based on an exclusive interview with Lawrence Broxmeyer MD


BULLET POINT SUMMARY

* COVID-19 Coronavirus is as much an ecological disaster as it is a medical one. Initially it appears to be a unique experience centered in Wuhan, China. It emanates from an environment of incinerated pig waste, airborne particles, and low vitamin D blood levels in winter, and weakened immune systems, particularly among smokers, drinkers and the elderly.
* It is believed both the Spanish flu of 1918 and the COVID-19 coronavirus began as zoonotic (animal to human) infections. Not from bats as first reported in the Wuhan COVID-19 outbreak, but rather from pigs, and pig waste.
* The 1918 Spanish flu pandemic began in the midst of an infectious pig slaughter of undiscovered cause, a few hundred miles from Camp Funston, what is Fort Riley today. * Similarly, the outbreak of the COVID-19 coronavirus outbreak began in the Wuhan, China area in the wake of a massive kill-off of pigs who were dying from African Swine Flu.
* Viral outbreaks arise in winter, but so does tuberculosis.
* Some types of mycobacteria do not have cell walls and can mimic the appearance of a virus under the microscope.
* Antibiotics cannot be used for viruses. If a virus, then why aren’t antiviral drugs working but antibiotics are?
COVID-19 coronavirus may just be a “passenger virus,” not the primary microbial organism that kills by filling the lower lungs with fluid.
* Both the current Wuhan COVID-19 coronavirus and tubercular mycobacteria do not tend to infect or cause serious disease in young children roughly 5-12 years of age.
* Fear of the COVID-19 coronavirus may be misplaced. More people are killed by Mycobacterium tuberculosis (1.7 million) in a year than the few who have been infected (~80,000) or have died (less than 2000) of the COVID-19 coronavirus.
* It is projected that the “COVID-19 Coronavirus” will peak worldwide in March and then return in a second but lesser peak in September, in accordance with Yang’s Wuhan study from 2004 to 2013, describing the annual TB surges in Wuhan, China.
* Saying the spread of the COVID-19 coronavirus is inevitable, a CDC (Centers for Disease Control) official advised Americans “brace themselves” and prepare to shut down public schools, avoid going to church, and self-quarantine their families. These onerous measures are for a virus that has infected just 53 Americans (Feb. 25), mostly among people who traveled recently to China.

Recognizing the possibility that some officials within the CDC are potential handmaidens to the drug and vaccine industry that would benefit from such scare tactics, President Trump decided to issue all governmental statements about COVID-19 from The White House.

After all, the CDC seemed to be about ready to bring down the entire U.S. economy and shutter small businesses that would never recover from even a short quarantine.

Doomsday virus?

Irresponsible and implausible predictions that COVID-19 coronavirus could produce symptomatic viral infection in 60% of the world’s population with 45 million deaths ̶ fueled by sensationalized news reports, were purely unfounded and certainly premature. Perhaps the only thing that could be considered beneficial about these dire prognostications was when one quick-thinking woman in China used the fear of the virus in a positive way, and feigned she was infected to foil a rapist.

In the beginning of the outbreak, you could have pulled from a widely-known advertising limerick used in Las Vegas: “Whatever coronavirus starts in China, stays in China.” Now we know that this is not quite the case. Airplanes facilitate its geographical spread, but person-to-person transmission remains static. Immune status may be a controlling factor in person-to-person transmission.

Maybe just a passenger virus

But what is the sense in panicking the world over a coronavirus that is 3 times less deadly than the SARS outbreak of 2003 (9.5% vs. 3.4%). And mind you, it has still not been proved that the COVID-19 coronavirus is anything more than a non-symptomatic, non-pathogenic “passenger virus,” being picked up diagnostically but not the primary underlying cause responsible for the Pandemic/Epidemic itself.

In fact, there is direct and indirect evidence that COVID-19 is NOT from a virus at all. The SARS coronavirus, often compared with the new COVID-19 coronavirus, was present only in about half of the SARS cases and antivirals such as ribavirin and oseltamivir (Tamiflu) were not working in SARS culture plates, nor were they very effective in clinics or hospitals either. That passenger viruses do exist has been abundantly documented, as when the HTLV-1 virus (human T-cell leukemia virus Type 1) was mistakenly attributed to being causal for AIDS. And so, the question which never went away looms: are the coronaviruses merely traveler viruses from a yet to be determined primary stealth cause?

Using bacteriocidals instead of antivirals

One recent US news headline said: “Scientists claim antibiotics already on the market could treat coronavirus – despite warnings from CDC and World Health Organization that antibiotics do nothing against viruses and overusing them fuels resistant microbes.

Fact of the matter is that antiviral medicines were not found to provide benefit in either the SARS or MERS strains of coronavirus either, which broke out in 2002 and 2012, respectively. Yet this habit of administering antivirals was stubbornly clung to in the treatment of the 138 Coronavirus-infected patients hospitalized in Wuhan, China described in Wang, et al’s recently published JAMA study.

On the other hand, all of these 138 patients, and most of the 99 patients in Chen et al’s current Lancet study received at least one antibiotic, some of which have significant anti-mycobacterial as well as antibacterial activity. (Mycobacteria are small rod-shaped bacteria, some varieties which have no cell wall and are difficult to distinguish from viruses; mycobacteria cause leprosy and tuberculosis, the latter usually emanating as lower respiratory tract infection that can mimic classic pneumonia, as observed in the SARS and COVID-19 coronavirus cases.)

Antibiotics have no effect against viruses. And although it is claimed that antibiotics are and were simply being used to quell “secondary” bacterial infections in the new pandemic, the fact is that antibiotics have proved universally to be of great help worldwide to the vast majority of novel coronavirus COVID-19 victims, with or without secondary infections.

Those who cannot remember the past are condemned to repeat it

While “experts” have been telling us to wash our hands, have they really been doing the factual research needed to compare COVID-19 to say the Great Pandemic of 1918? Dr. Lawrence Broxmeyer MD, whose writings were previously published in the highly ranked The Journal of Infectious Diseases, doesn’t seem to think so. And his views, as expressed in an upcoming publication, aren’t alone.

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50% rule observed