hostile humans and cannibalistic creatures infected by a mutated strain of the Cordyceps fungus

Who lives in a fungus? The diversity, origins and functions of fungal endobacteria living in Mucoromycota

Since the discovery of antibiotics, fungi and bacteria have been assumed to interact antagonistically, but new ideas have emerged in microbial studies: as elements of the same microbiota, fungi and bacteria may interact non-antagonistically...

The most intimate interaction takes place when bacteria live inside the fungal cells as endobacteria. Irrespective of their genetic and functional diversity, fungus-associated bacterial communities constitute a novel type of microbiota, the fungal microbiota...Numerous recent studies have identified endobacteria in several fungal lineages...

...One of the better-known example involving a betaproteobacterium is the rice pathogenic fungus Rhizopus microsporus (Mucoromycotina), the causal agent of rice seedling blight, whose pathogenicity is due to the presence of the endosymbiont Burkholderia rhizoxinica (Partida-Martinez and Hertweck, 2005). Indeed, B. rhizoxinica produces the rhizoxin used as a virulence factor by the fungus (Partida-Martinez and Hertweck, 2007). Furthermore, this facultative endobacterium affects the vegetative reproduction of the fungal host, controlling the formation of sporangia and spores (Partida-Martinez et al., 2007).
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The presence of the itch, misdiagnosed and unknown to the peasant class that eats the blight: [link to onlinelibrary.wiley.com (secure)]

Connections of bacteria called singular virus, as a contagious prostate cancer became AIDS, due to the bacteria getting into the bloodstream of gjeys from buttsex: [link to www.bitchute.com (secure)] the same is here: [link to www.sciencedirect.com (secure)]

[link to www.bitchute.com (secure)]

Bacteria - Candidatus Neoehrlichia mikurensis is an emerging tick-borne pathogen causing a systemic inflammatory syndrome mostly in persons with underlying hematologic or autoimmune diseases. As it is neither well-known nor well-recognized, it might be misdiagnosed as recurrence of the underlying disease or as an unrelated arteriosclerotic vascular event. The pathogen is transmitted by hard ticks of the genus Ixodes and is closely associated with rodents in which transplacental transmission occurs.
[link to www.science.gov (secure)]

[link to www.npr.org (secure)]

Covid blamed. This seems to be related to diabetes. Steroids blamed.
Sometimes eyes and jawbones have to be taken out.

So thousands have Covid, Diabetes and caught a fungal infection of Mycormycosis.


Yeah, it could happen.

Figure 13
Cryptococcosis of lung in patient with AIDS. Histopathology of lung shows widened alveolar septum containing a few inflammatory cells and numerous yeasts of Cryptococcus neoformans CDC/Dr. Edwin P. Ewing, Jr. [email protected]

Figure 14
Cryptococcosis of lung in patient with AIDS. Mucicarmine stain. Histopathology of lung shows widened alveolar septum containing a few inflammatory cells and numerous yeasts of Cryptococcus neoformans. The inner layer of the yeast capsule stains red. CDC/Dr. Edwin P. Ewing, Jr. [email protected]
[link to www.microbiologybook.org (secure)]

add: [link to market-ticker.org (secure)]

Thread: Jurgen Konings, the RAMBO of BELGIUM found DEAD



The AIDS connection again...

Marc Van Ranst
He is also the director of the AIDS reference laboratory and of the national coronavirus and rotavirus reference laboratories. On 1 May 2007, he was appointed as Interministerial comissionar by the Belgian federal government to prepare Belgium for an influenza pandemic. In 2009-10 he was an interministerial commissary for crisis management during the Mexican flu pandemic[1] In 2020, during the COVID-19 pandemic, Marc Van Ranst became a member both of the Belgian 'Risk Assessment Group' (RAG), which analyses the risks of coronavirus SARS-CoV-2.
[link to en.wikipedia.org (secure)]

Breaking: Jürgen Conings found dead in Hoge Kempen National Park
[link to www.brusselstimes.com (secure)]

Informed consent disclosure to vaccine trial subjects of risk of COVID‐19 vaccines worsening clinical disease
Timothy Cardozocorresponding author 1 and Ronald Veazey 2
Author information Copyright and License information Disclaimer
This article has been cited by other articles in PMC.
Associated Data

Data Availability Statement

Abstract
Aims of the study

Patient comprehension is a critical part of meeting medical ethics standards of informed consent in study designs. The aim of the study was to determine if sufficient literature exists to require clinicians to disclose the specific risk that COVID‐19 vaccines could worsen disease upon exposure to challenge or circulating virus.
Methods used to conduct the study

Published literature was reviewed to identify preclinical and clinical evidence that COVID‐19 vaccines could worsen disease upon exposure to challenge or circulating virus. Clinical trial protocols for COVID‐19 vaccines were reviewed to determine if risks were properly disclosed.
Results of the study

COVID‐19 vaccines designed to elicit neutralising antibodies may sensitise vaccine recipients to more severe disease than if they were not vaccinated. Vaccines for SARS, MERS and RSV have never been approved, and the data generated in the development and testing of these vaccines suggest a serious mechanistic concern: that vaccines designed empirically using the traditional approach (consisting of the unmodified or minimally modified coronavirus viral spike to elicit neutralising antibodies), be they composed of protein, viral vector, DNA or RNA and irrespective of delivery method, may worsen COVID‐19 disease via antibody‐dependent enhancement (ADE). This risk is sufficiently obscured in clinical trial protocols and consent forms for ongoing COVID‐19 vaccine trials that adequate patient comprehension of this risk is unlikely to occur, obviating truly informed consent by subjects in these trials.
Conclusions drawn from the study and clinical implications

The specific and significant COVID‐19 risk of ADE should have been and should be prominently and independently disclosed to research subjects currently in vaccine trials, as well as those being recruited for the trials and future patients after vaccine approval, in order to meet the medical ethics standard of patient comprehension for informed consent.
1. THE RISK OF ADE IN COVID‐19 VACCINES IS NON‐THEORETICAL AND COMPELLING

Vaccine‐elicited enhancement of disease was previously observed in human subjects with vaccines for respiratory syncytial virus (RSV), dengue virus and measles. 1 Vaccine‐elicited enhancement of disease was also observed with the SARS and MERS viruses and with feline coronavirus, which are closely related to SARS‐CoV‐2, the causative pathogen of COVID‐19 disease.
[link to www.ncbi.nlm.nih.gov (secure)]

I think it’s both - a parasitic virus.
[link to pubmed.ncbi.nlm.nih.gov (secure)]