what i see is 2 big points.
1> on surfaces the virus break down quickly. stays viable on plastic the longest. but we still do not know the time line on this. and there is a variable to it see point 2.
2> the virus randomly creates infectious cells,probly wrong term. it also is random on how many a person will make. it also seems to b random on the virus making infectious cells,again probly wrong word, once a person is infected.
from that we can take a few things. who and how anyone is infected or becomes sick is totally random. 1 person might b a carrier to infect others and have nothing but a cold. another might get sick and die in hours or days. and there is a lot of gray zone between the 2.
put another way. 1 person might make 1/1 million cells that infect another. another person might b 50/50. 1 might get sick as we have seen with hot symptons and the tiredness that comes with it. another might need a vent and b dead in a month. where up til now, we were under the impression there was a DNA genetic componet to it,it is now all random.
let me cringe at this and post more in a bit.
Quoting: JAZZz50 A) I wrote back when Mrs Doom was doing the research on how long the virus was capable of living on surfaces - and Wordsie is correct - I do quarantine all our paper mail. And clean EVRYTHING else down with hypochlorous acid before it gets into the inner sanctum. Us included (Halo fire and rescue shower is perfect. Works wonders).
7 days for plastic is now the accepted numbers. A whole freaking week as long as it (sarscov2) is exposed to air and changes in humidity. If it is inside a protected atmosphere with slight moisture available AND stable temps - Mrs Doom et al did find that they had 15 days as their max recommended sanitization time for plastic covered goods.
Here is another teams early efforts and findings. Look closely as the humidity / weather play a MASSIVE part.
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link to www.ncbi.nlm.nih.gov (secure)]
B) Burst. I have sat through the slides and the best way I can put this is that the virus has been designed to evade defences AND create itself an environment where it can thrive, multiply to the point of spread, and then create a catastrophic event inside the host after a long dwell time.
Every aspect points directly to being designed this way.
The 'random' is NOT random at all. It is a calculated pathway; if event A happens, response B will take place. If event B happens, response C & D will take place. The design is purposefully creating what appears to be a random virus with no discernable pattern - Until you take the effected in as seperate cohorts; examples being C1, 14-19, White, Eurasian DNA. Rising testosterone. C2, 23-38, White, Eurasian, peak testosterone and peak cardiac health... So on and so forth. Break it down, take cells that have undergone SPECIFIC PATTERNS OF GROWTH AND / OR DAMAGE, and you see that this bastard fucker is a targeted mother fucker of a payload.
Break it down. Get the figures.
RACE
SEX
CELLULAR MATURITY
PEAK CELLULAR ACTIVITY
Testosterone.
DNA AND CELLULAR MARKERS THAT SIGNAL POOR HEALTH / metabolic outcomes.
Start cutting the deaths AND THE ADVERSE REACTIONS and you will see, stark and clear, just how this fucker was and is tailored to cut swathes through specific groups. We are just being bamboozled by the media to look the other way.
Then the 'random' burst is NOT random at all.
